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Part of the NATO ASI Series book series (volume 21). Abstract. In: Dowdall MJ, Hawthorne JN (eds) Cellular and molecular basis of cholinergic function
Part of the NATO ASI Series book series (volume 21). ATP is stored in cholinergic synaptic vesicles of the peripheral (Dowdall et al 1974; Volknandt and Zimmermann 1986) and central (Richardson and Brown 1987) nervous system, in adrenergic vesicles, in chromaffin granules and also in serotonin containing granules of blood platelets (ref. Zimmermann 1982). In: Dowdall MJ, Hawthorne JN (eds) Cellular and molecular basis of cholinergic function. Ellis Horwood, Chichester, and VCH, Weinheim, pp 444–453Google Scholar.
Cellular and Molecular Basis of Cholinergic Function. Ellis Horwood Series in Biomedicine. (1 March, 1988) Free. Multiple Sclerosis, Immunological, Diagnostic and therapeutic aspects. Current Problems in Neurology 3. Computer Tomographic Imaging and Anatomic Correlation of the Human Brain (1 March, 1988) Free.
PubMed comprises more than 30 million citations for biomedical literature from MEDLINE, life science journals, and online books. More recently, a fascinating new role of these enzymes has been unveiled. The activating deiodinase (D2) and the inactivating deiodinase (D3) can locally increase or decrease thyroid hormone signaling in a tissue- and temporal-specific fashion, independent of changes in thyroid hormone serum concentrations.
Ellis Horwood series in biomedicine, 0930-3367. Personal Name: Hawthorne, J. N. (John Nigel). Uniform Title: Ellis Horwood series in biomedicine. Rubrics: Cholinergic mechanisms Acetylcholine Receptors Physiological effect Receptors, Cholinergic physiology.
This book contains 105 selections. Some of the titles are: Functional correlates of brain nicotine receptors; Muscarinic receptor subclasses; Cholinergic innervation and levels of nerve growth factor and its mRNA in the central nervous system; Developmentally regulated neurontrophic activities of Torpedo electric organ tissue; and Association of a regulatory peptide with cholinergic neurons.
In: Cellular and Molecular Basis of Cholinergic Function. Hawthorne, ed. Ellis Harwood, Chichester. Sussex) UK, p. 41-346, 1987. Liscovitch, Mauron, . Richardson, . Phosphatidylcholine as a precursor of choline for acetylcholine.
In: Dowdall MJ, Hawthorne JN, eds. Cellular and Molecular Basis of Cholinergic Function. Biochemical assessment of serotonergic and cholinergic dysfunction and cerebral atrophy in Alzheimer's disease. J Neurochem 1983; 41: 266-272. Chichester: Ellis Horwood, 1987: 858-868. 43. Arendt T, Bigl V. Alzheimer's plaques and cortical cholinergic innervation. Neurology 1986; 17: 277-279. 46. Perry EK, Marshall EF, Blessed G. Decreased imipramine binding in the brains of patients with depression.
I am in terested in understanding the role of transcription factors that modulate cellular function in such pathway.
Drug-induced molecular and cellular plasticity may result in tolerance and/or sensitization (Hyman et a. 2006; Koob and Le Moal, 2008;Stewart and Badiani, 1993). Whereas tolerance refers to a decrease in the effectiveness of a given drug, sensitization involves a progressive increase in its effect ( Kalivas and Stewart, 1991;Nestler and Aghajanian, 1997;Stewart and Badiani, 1993). I am in terested in understanding the role of transcription factors that modulate cellular function in such pathway.
Twenty-eight comprehensive chapters, authored by internationally recognized leaders in the field, discuss the biophysical, physiological, and molecular characteristics of cell-to-cell communication via gap junctions.